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These data suggest the presence of two unique and very distinct neuronal circuitries within the AP and highlight the hypothesis of a local inhibitory network within the caudal hindbrain that could be exploited via GIPR activation to reduce hindbrain GLP-1R–mediated emesis and nausea (Fig

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Butyrate limits the replication of porcine epidemic diarrhea virus in intestine epithelial cells by enhancing GPR43-mediated IFN-III production

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