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A PPARβ/δ inhibitor, GSK0600, was employed at a concentration of 0.2 µM to validate the critical role of the PPARβ/δ-β-catenin-TCF-4 pathway in mediating GLP-1 release in response to C3G treatment
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Quantity:2 Bottles (60 Day Supply)
Prolonged hyperglycemia accelerates this glycation process, resulting in the accumulation of AGEs both endogenously through metabolic pathways and exogenously from dietary sources
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